Mitochondria of HPUs particularly susceptible to metal contamination
A study published in August 2019 in the journal “OM & Ernährung” showed a correlation between metal exposure and mitochondrial function in newly diagnosed HPUs (Huesker, Thiem and Assheuer, OM & Ernährung 2019; SH14: 26-28).
The researchers used laboratory values collected from newly diagnosed HPU patients at the Institute for Medical Diagnostics (IMD) in Berlin. The exciting discovery:
The daily excretion of hydroxy-hemopryrrollactam (HPL, measured at KEAC, Netherlands) correlates with the arsenic concentration in the blood of newly diagnosed HPUs.
In other words, the higher the HPL value in the HPU test, the more lead the scientists found in the blood of the patient concerned.
Another observation stood out: The more lead and arsenic the HPU patients had in their blood, the less intracellular ATP was detectable. Intracellular ATP is a laboratory parameter that provides information on the function of the mitochondria. Low ATP values indicate poor mitochondrial function. HPUs seem to be particularly sensitive to arsenic and lead exposure.
Tip for therapists:
HPU therapists should therefore detect chronic metal contamination and eliminate the sources (at the IMD in Berlin as multi-element analysis in the blood analysis 103 or 271; as targeted individual analysis in EDTA whole blood also as a SHI service). ATP is a meaningful biomarker when metal-associated mitochondrial damage in HPUs needs to be diagsnoted (IMD Analysis 91, sham “Special Immunology”).